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Extensive research has unveiled intricate interactions and mechanisms between the gut and lung infections, encompassing bacterial translocation, short-chain fatty acids, and recognition receptors for pathogen-associated molecular patterns

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Research has demonstrated that glutamate dehydrogenase (GLUD1) is expressed at a high level in lung adenocarcinoma cells, and lysine 503 (K503) has been identified as the primary ubiquitination site of GLUD1